In an article entitled, “Glutamate receptor-like genes mediate leaf –to-leaf wound signaling,” published on the 22nd August, ‘13 issue of Nature, (500: 422–426), Seyed AR Mousavi and four of his associates affiliated to the Department of Plant Molecular Biology, University of Lausanne, School of Pharmaceutical Sciences, University of Geneva and Department of Pharmacology and Toxicology, University of Lausanne report that wounded leaves communicate their  condition of injury to one another through  a process not understood fully. According to the authors, long-distance signaling is the probable cause for stimulation of  the distal production of jasmonates – the chemicals that regulate  strongly defense responses. Using non-invasive electrodes the authors  mapped surface potential changes in Arabidopsis thaliana after wounding leaf eight. They discovered  that membrane depolarization is correlated with jasmonate signaling domains in intact leaves. Furthermore, they observed that current injection lead to jasmonoyl-isoleucine accumulation, resulting in a transcriptome enriched in RNAs encoding key jasmonate signaling regulators. From among 34 screened membrane protein mutant lines, mutations in several taxa 3 GLUTAMATE RECEPTOR-LIKE genes (GLRs 3.2, 3.3 and 3.6) were found to reduce wound-induced surface potential changes. Jasmonate-response gene expression in leaves distal to wounds was reduced in a glr3.3 glr3.6 double mutants. This work provides a possible clue explaining  mechanisms of long-distance wound signaling in plants.